![]() In the low dose (1 mg) dexamethasone suppression test, serum cortisol level was suppressed to the normal range. Levels of catecholamine and its metabolites in 24-h urine were normal. The patient was detected for multiple endocrine neoplasia syndromes. Aldosterone level was measured after normalization of his serum potassium level (result: 26.9 ng/dl). Calcium level in 24-h urine: 251 mg/day, inorganic P: 88 mg/day, and K + value in spot urine: 66 mmol/l. The other systemic and laboratory findings were the following: complete blood count and thyroid function tests were normal: serum Ca level: 10.9 mg/dl (8.8–10.2), inorganic P: 2.4 mg/dl (2.7–4.5), Mg: 2.3(1.6–2.6), albumine: 3.9 g/dl, parathormone (PTH): 200 pg/ml (12–88), Na: 138 mmol/l, K +: 2.8 mmol/l, glucose: 85 mg/dl, creatinine: 0.84 mg/dl, alanine transaminase: 24 U/l, white blood cells 8 ML, hemoglobin 14 g/dl, platelet 219 C-reactive protein 0.4 mg/dl (0–0.8), alkaline phosphatase 61 U/l (38–126), 25-OH vitamin D3 22 ng/ml (14–66). His general clinical appearance was good. On his physical examination: blood pressure-150/90 mmHg, pulse −74 per minute, rhythmic. ![]() There is no other significant clinical data in his medical history. His third child is 2 years old and living with the respiratory support equipment. ![]() One of them died at the age of 13 months, while the other 9 months of age. His three children were diagnosed with West Syndrome. His father died at 47 years of age due to biliary tract cancer. In his family history, none of his first degree relatives had hypertension. Due to the complications, like hypopotassemia this medicine was replaced with calcium channel blockers. History revealed, that he took ace inhibitor in combination with thiazide diuretic for hypertension. ![]() Before entering our clinic, hypercalcemia has been detected in another medical center. A 38-year-old Turkish male was admitted to our outpatient clinic for hypercalcemia. ![]()
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